Ribosomal Remodeling and Translational Suppression in the Post-Arrest Heart
Background: Cardiac arrest (CA) is a leading cause of death and disability in the United States, and no pharmacologic therapies improve post-arrest outcomes. Acute myocardial dysfunction is a highly prevalent CA complication that contributes to post-arrest mortality, yet the molecular mechanisms that drive this dysfunction remain incompletely understood. Prior work in our laboratory has suggested…
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