Study identifies key driver of inflammation, opening door to potential new treatments
Researchers at Johns Hopkins Medicine have uncovered a critical biological mechanism that helps explain how inflammation is triggered and sustained in a wide range of diseases—highlighting a promising avenue for future therapeutic development.
Researchers at Johns Hopkins Medicine have identified human resistin as a crucial factor in the onset and persistence of inflammation, a condition linked to numerous health issues such as cardiovascular disease, diabetes, and autoimmune disorders. Published in PLOS One, the study reveals that resistin acts as a "switch," initiating and activating immune cells known as macrophages, which in turn triggers a series of events leading to inflammation.
This discovery opens new avenues for developing targeted therapies to interrupt harmful inflammatory responses.
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